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Although ectopic activation of mTORC1 in peripheral tissues might be attained by administering a substantial dose of exogenous leucine, mTORC1 signaling while in the CNS stays refractory probably due to fast turnover and buffering of leucine during the brain18,19. The discovery of Sestrin1 and 2 as a specific leucine sensor https://ammonj665xiu9.blogunteer.com/profile


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